The sacroiliac joint is the least mobile major joint in the body and one of the most heavily ligamented. Understanding that combination explains most of what follows: why it rarely goes “out”, why the ligaments behind it are a pain source in their own right, and why the single most useful question is not whether the SIJ hurts but which part of the SIJ complex hurts.

What the joint actually is

The sacroiliac joint connects the sacrum — the wedge of fused vertebrae at the base of the spine — to the ilium on each side. It is the only structural link between the spine and the legs, so every step, every stair and every lift passes load through it.

It is a hybrid joint. The lower, front portion is a true synovial joint with cartilage-covered surfaces. The upper, rear portion is not a joint surface at all but a syndesmosis: a dense mass of interosseous ligament packing the space between the two bones. That is unusual, and it is the single anatomical fact that most changes how the joint is assessed and treated.

Movement is small. The joint is not designed to glide; it is designed to transfer force while deforming very little. This is why the popular idea of a sacroiliac joint that slips out of place and needs putting back is not supported by what is measurable at the joint.

Form closure and force closure

Two mechanisms hold the pelvis together under load, described by Vleeming and colleagues as form closure and force closure.

Form closure is the stability that comes from the shape of the parts. The sacrum is wedged between the ilia, the joint surfaces carry matching ridges and grooves that produce a high coefficient of friction, and the binding ligaments are among the strongest in the body. A perfectly form-closed joint would need no muscular help at all — but it would also be immobile.

Force closure is the extra compression supplied by ligaments, fascia and muscle acting across the joint, plus the ground reaction force. It is what allows a joint with imperfect form closure to still tolerate a vertical load. Force closure depends on sacral nutation — a small forward rotation of the sacrum relative to the ilia — which tightens the dorsal ligaments and presses the posterior ilia together in anticipation of loading.

This is a model, not a measurement. It is useful because it predicts why some people improve with better muscular control and a period of load management, and why others do not. It is not a licence to tell someone their pelvis is unstable — see lumbopelvic instability for where that idea holds up and where it does not.

The ligaments, one by one

Structure Where it runs What it does Why it matters clinically
Interosseous sacroiliac ligamentFills the space between sacrum and ilium above and behind the synovial jointThe principal binding structure; the syndesmotic part of the jointThe target of the CT-guided dextrose protocol described by Cusi and colleagues in Sydney. Not reachable by an injection into the joint cavity.
Long dorsal sacroiliac ligamentFrom the posterior superior iliac spine down to the lateral sacrumTightens in counternutation, slackens in nutation — the reverse of the other dorsal ligamentsDirectly palpable just below the PSIS. Tender in a high proportion of women with peripartum pelvic pain, and a plausible pain generator on its own.
Sacrotuberous ligamentSacrum and coccyx to the ischial tuberosityResists sacral nutation — the reverse of the long dorsal ligamentCadaveric loading showed that tension in this ligament increases tension in the long dorsal ligament, so the two cannot be considered separately.
Sacrospinous ligamentLateral sacrum and coccyx to the ischial spineResists nutation; forms the border of the greater and lesser sciatic foraminaSits in the deep gluteal space. Where it calcifies it can, rarely, contribute to sciatic-type pain.
Anterior sacroiliac ligamentAcross the front of the jointThin compared with the posterior structuresRelatively weak; the front of the joint is not where the ligamentous strength is.
Thoracolumbar fascia and its attachmentsBroad sheet linking latissimus dorsi, erector spinae, gluteus maximus and the sacrumTransmits force between the arms, trunk and legs across the pelvisVleeming's cadaveric loading work showed traction on these structures measurably changes tension in the long dorsal ligament. This is the anatomical basis for treating the pelvis as part of a chain rather than in isolation.

Intra-articular and extra-articular pain are roughly equally common

This is the finding that reorganises everything. The 2025 multispecialty consensus guidelines concluded that the prevalence of intra-articular and extra-articular pathology in SIJ complex pain is comparable — pain arises from the ligamentous and posterior structures about as often as it arises from inside the joint.

The guidelines went further. They found the evidence slightly stronger for extra-articular corticosteroid injection than for intra-articular injection in providing short-term relief, and they concluded that intra-articular injections have diagnostic validity for intra-articular pain but not for extra-articular pain. One participating society abstained on the extra-articular statement; the rest reached consensus.

The practical consequence: an injection into the joint cavity that does not relieve the pain has not ruled out the SIJ complex as the source. It has ruled out one half of it. That is a distinction the older literature — and much of what patients read online — does not make.

Why the term is now 'SIJ complex pain'

  • The joint cavity, the interosseous ligament, the long dorsal ligament and the surrounding posterior structures are treated as one anatomical complex rather than as a joint plus some soft tissue.
  • A test, a block or a procedure that addresses one component says little about the others.
  • Roughly 15 to 30 per cent of people with axial pain predominantly below the L5 level have pain arising from this complex.

Nerve supply — and why nobody agrees on it

The posterior SIJ complex is supplied by the lateral branches of the sacral dorsal rami, usually S1 to S3 and often with a contribution from the L5 dorsal ramus. These branches form a variable network across the back of the sacrum — a posterior sacral network with no reliable, repeatable topography from person to person.

That variability is the reason nerve-targeted procedures at this joint are technically demanding and why the guideline evidence favours larger, more aggressive lesions over precise small ones. A cadaveric study by Roberts and colleagues found that needles placed along the lateral sacral crest under ultrasound would capture on average 93 per cent of the posterior sacral network if a strip lesion were created between them — an argument for the strip approach rather than for point targeting.

The front of the joint has a separate and even less consistent supply from the ventral rami. No current procedure reliably denervates it, which is part of why nerve ablation at this joint is described as a treatment for extra-articular rather than intra-articular pain.

What this anatomy rules out

It rules out the idea that the sacroiliac joint moves in and out of position. Measured motion at the joint is very small, and no palpation test has been shown to detect it reliably — a point developed on the diagnosis page.

It rules out the idea that a single injection anywhere near the joint treats the whole complex. The joint cavity and the interosseous ligament are separate compartments requiring separate approaches.

And it rules out the idea that the pelvis can be assessed in isolation. The long dorsal ligament changes tension when the latissimus dorsi, erector spinae, gluteus maximus or sacrotuberous ligament are loaded. Anything that alters hip, trunk or hamstring function alters what this joint experiences.

Related on this site

Common questions

Can the sacroiliac joint go out of place?
Not in the way the phrase suggests. Movement at the joint is very small and is resisted by some of the strongest ligaments in the body. Systematic review of the palpation tests used to detect sacroiliac positional faults found agreement between examiners ranging from no better than chance to moderate at best, and no study has ever demonstrated that these tests measure what they claim to measure. Pain in this region is real; the explanation that the joint has slipped is not supported.
How much does the sacroiliac joint actually move?
Very little. It is a hybrid joint — a small synovial portion at the front and below, and a syndesmosis packed with interosseous ligament above and behind. It is built to transfer load with minimal deformation rather than to move through a range.
Which SIJ ligament is most often painful?
The long dorsal sacroiliac ligament is the one most consistently implicated, particularly after pregnancy. Vleeming and colleagues found tenderness on standardised palpation of this ligament in 76 per cent of women meeting strict criteria for peripartum pelvic pain, rising to 86 per cent in those also positive on the active straight leg raise and posterior pelvic pain provocation tests. That is a sensitivity figure, not a specificity figure — a tender ligament supports the suspicion but does not confirm the diagnosis.
Is pain from the ligaments different from pain from inside the joint?
They cannot be told apart by where they hurt or by examination alone. The 2025 consensus guidelines found that a battery of physical examination tests has reasonable sensitivity for intra-articular pain but does not identify extra-articular pain at all, and that an injection into the joint has no diagnostic validity for the extra-articular structures. This is why the distinction is made by which structure is targeted rather than by symptom pattern.

Evidence reviewed

Every clinical statement on this page traces to one of the sources below, including the ones that point the other way. Where a study is small, unblinded or authored by people with a commercial interest in the result, that is noted alongside it rather than left out.

Last reviewed 2026-09-04. This is general information about a condition and its treatments, not personal medical advice, and it is no substitute for assessment by a clinician who has examined you.

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