Frozen shoulder is a painful stiffening condition of the shoulder joint capsule. It often causes marked loss of movement and can affect sleep, dressing, reaching and exercise.
Frozen shoulder, or adhesive capsulitis, causes pain and progressive restriction of shoulder movement. It can occur without a clear trigger or after injury, surgery or prolonged guarding. It is more common in people with diabetes or thyroid disease.
Assessment is important because frozen shoulder can be confused with rotator cuff pain, arthritis or referred pain from the neck. The key finding is restriction of both active and passive shoulder movement.
Management depends on the phase. During the painful phase, aggressive stretching can flare symptoms. Treatment often focuses on pain control, sleep, gentle mobility and maintaining function. As pain settles, progressive mobility and strengthening become more useful.
Evidence-informed treatment summary
How our treatment options may fit for Frozen Shoulder
The options below include the treatments offered at The Back Pain Doctor. Listing a treatment does not mean it is recommended for this condition. The evidence, likely benefit and role of each option are considered against the diagnosis, examination findings, imaging where appropriate, patient goals, risks, cost and alternatives.
Foundation
Diagnosis, education and progressive rehabilitation
This is the starting point for most musculoskeletal conditions.
The priority is to identify the likely pain generator, explain the condition clearly, modify aggravating load and build a realistic plan to restore strength, movement and confidence.
Evidence is condition-specific; it is not a universal pain treatment.
Shockwave is best framed as an adjunct where the diagnosis fits. It is generally more established for selected tendon and plantar heel pain presentations than for many joint or nerve conditions.
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→ Evidence varies substantially by condition, tissue and preparation method.
PRP may be discussed in selected tendon or joint presentations. It should not be presented as a guaranteed regenerative treatment, and uncertainty, cost and alternatives should be discussed.
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→ May provide symptom relief when the pain generator is a joint, bursa, tendon sheath or other defined irritable structure.
Injection treatment may be discussed when pain is limiting sleep, function or rehabilitation. The expected benefit is usually symptom control, not tissue regeneration.
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→ Clinical evidence is still developing and guideline support is limited.
EMTT may be discussed as an adjunct in selected presentations, but should be presented with clear uncertainty and never as a replacement for diagnosis, load management or rehabilitation.
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→ Evidence is condition-specific and generally less established than exercise-based care.
Prolotherapy may be considered in carefully selected chronic ligament, tendon or joint-related pain presentations, but it is not a first-line treatment.
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→ Most relevant when focal myofascial pain is a clear contributor.
Trigger point treatment may reduce pain from focal muscle spasm or myofascial tenderness. It should be paired with movement restoration, strength work and recurrence prevention.
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→ Relevant only when the history and examination support nerve irritation or entrapment.
Nerve-focused treatment may be discussed when there is a plausible peripheral nerve pain generator. Progressive weakness, major neurological deficit or red flags require a different pathway.
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This is general information only. Suitability is assessed individually. Treatments with limited or condition-dependent evidence may still be discussed, but only with clear explanation of uncertainty, expected benefit, risks, cost and alternatives. Red flags, progressive neurological symptoms or suspected serious pathology require a different pathway.